Immunization with advanced glycation end products modified low density lipoprotein inhibits atherosclerosis progression in diabetic apoE and LDLR null mice

BACKGROUND: Diabetes accelerates atherosclerosis through undefined molecular mechanisms. Hyperglycemia induces formation of advanced glycation end product (AGE)-modified low-density lipoprotein (LDL). Anti-AGE-LDL autoantibodies favor atherosclerosis (AS) progression in humans, while anti oxidized L...

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Sparad:
Bibliografiska uppgifter
I publikationen:Cardiovasc Diabetol
Huvudupphovsmän: Zhu, Lin, He, Zhiqing, Wu, Feng, Ding, Ru, Jiang, Qixia, Zhang, Jiayou, Fan, Min, Wang, Xing, Eva, Bengtsson, Jan, Nilsson, Liang, Chun, Wu, Zonggui
Materialtyp: Artigo
Språk:Inglês
Publicerad: BioMed Central 2014
Ämnen:
Original Investigation
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC4234834/
https://ncbi.nlm.nih.gov/pubmed/25391642
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12933-014-0151-6
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