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Repression of the Arf tumor suppressor by E2F3 is required for normal cell cycle kinetics
Tumor development is dependent upon the inactivation of two key tumor-suppressor networks, p16(Ink4a)–cycD/cdk4–pRB–E2F and p19(Arf)–mdm2–p53, that regulate cellular proliferation and the tumor surveillance response. These networks are known to intersect with one another, but the mechanisms are poor...
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| Publicado no: | Genes Dev |
|---|---|
| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Cold Spring Harbor Laboratory Press
2004
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC423192/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15175242/ https://ncbi.nlm.nih.govhttps://doi.org/10.1101/gad.1196704 |
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