Immobilization contributes to exaggerated neuropeptide signaling, inflammatory changes, and nociceptive sensitization after fracture in rats

Lignende værker

• Facilitated spinal neuropeptide signaling and upregulated inflammatory mediator expression contribute to postfracture nociceptive sensitization
  af: Shi, Xiaoyou, et al.
  Udgivet: (2015)
• Substance P spinal signaling induces glial activation and nociceptive sensitization after fracture
  af: Li, Wen-Wu, et al.
  Udgivet: (2015)
• Neuropeptides Contribute to Peripheral Nociceptive Sensitization by Regulating Interleukin-1 Beta Production in Keratinocytes
  af: Shi, Xiaoyou, et al.
  Udgivet: (2011)
• TNF signaling contributes to the development of nociceptive sensitization in a tibia fracture model of complex regional pain syndrome type I
  af: Sabsovich, Ilya, et al.
  Udgivet: (2007)
• Neuropeptide deficient mice have attenuated nociceptive, vascular, and inflammatory changes in a tibia fracture model of complex regional pain syndrome
  af: Guo, Tian-Zhi, et al.
  Udgivet: (2012)