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Misfolding of vWF to Pathologically Disordered Conformations Impacts the Severity of von Willebrand Disease

The primary hemostatic von Willebrand factor (vWF) functions to sequester platelets from rheological blood flow and mediates their adhesion to damaged subendothelium at sites of vascular injury. We have surveyed the effect of 16 disease-causing mutations identified in patients diagnosed with the ble...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: Tischer, Alexander, Madde, Pranathi, Moon-Tasson, Laurie, Auton, Matthew
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: The Biophysical Society 2014
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4156683/
https://ncbi.nlm.nih.gov/pubmed/25185554
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bpj.2014.07.026
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