Upregulation of NHE1 protein expression enables glioblastoma cells to escape TMZ-mediated toxicity via increased H(+) extrusion, cell migration and survival

Sodium–hydrogen exchanger isoform 1 (NHE1) plays a role in survival and migration/invasion of several cancers and is an emerging new therapeutic target. However, the role of NHE1 in glioblastoma and the interaction of NHE1 expression and function in glioblastoma cells with cytotoxic temozolomide (TM...

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Bibliografiska uppgifter
Huvudupphovsmän: Cong, Damin, Zhu, Wen, Shi, Yejie, Pointer, Kelli B., Clark, Paul A., Shen, Hongmei, Kuo, John S., Hu, Shaoshan, Sun, Dandan
Materialtyp: Artigo
Språk:Inglês
Publicerad: Oxford University Press 2014
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Original Manuscript
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC4146414/
https://ncbi.nlm.nih.gov/pubmed/24717311
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/carcin/bgu089
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