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MeCP2 post-translational modifications: a mechanism to control its involvement in synaptic plasticity and homeostasis?
Although Rett syndrome (RTT) represents one of the most frequent forms of severe intellectual disability in females worldwide, we still have an inadequate knowledge of the many roles played by MeCP2 (whose mutations are responsible for most cases of RTT) and their relevance for RTT pathobiology. Sev...
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| 主要な著者: | , , , , , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Frontiers Media S.A.
2014
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4131190/ https://ncbi.nlm.nih.gov/pubmed/25165434 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fncel.2014.00236 |
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