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MeCP2 post-translational modifications: a mechanism to control its involvement in synaptic plasticity and homeostasis?
Although Rett syndrome (RTT) represents one of the most frequent forms of severe intellectual disability in females worldwide, we still have an inadequate knowledge of the many roles played by MeCP2 (whose mutations are responsible for most cases of RTT) and their relevance for RTT pathobiology. Sev...
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| Autors principals: | , , , , , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Frontiers Media S.A.
2014
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4131190/ https://ncbi.nlm.nih.gov/pubmed/25165434 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fncel.2014.00236 |
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