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MeCP2 post-translational modifications: a mechanism to control its involvement in synaptic plasticity and homeostasis?

Although Rett syndrome (RTT) represents one of the most frequent forms of severe intellectual disability in females worldwide, we still have an inadequate knowledge of the many roles played by MeCP2 (whose mutations are responsible for most cases of RTT) and their relevance for RTT pathobiology. Sev...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: Bellini, Elisa, Pavesi, Giulio, Barbiero, Isabella, Bergo, Anna, Chandola, Chetan, Nawaz, Mohammad S., Rusconi, Laura, Stefanelli, Gilda, Strollo, Marta, Valente, Maria M., Kilstrup-Nielsen, Charlotte, Landsberger, Nicoletta
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Frontiers Media S.A. 2014
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4131190/
https://ncbi.nlm.nih.gov/pubmed/25165434
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fncel.2014.00236
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