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p53 controls both the G2/M and the G1 cell cycle checkpoints and mediates reversible growth arrest in human fibroblasts.

Increased expression of wild-type p53 in response to DNA damage arrests cells late in the G1 stage of the cell cycle by stimulating the synthesis of inhibitors of cyclin-dependent kinases, such as p21/WAF1. To study the effects of p53 without the complication of DNA damage, we used tetracycline to r...

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Udgivet i:Proc Natl Acad Sci U S A
Principais autores: Agarwal, M L, Agarwal, A, Taylor, W R, Stark, G R
Format: Artigo
Sprog:Inglês
Udgivet: National Academy of Sciences 1995
Fag:
Research Article
Online adgang:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC41183/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7667317/
https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.92.18.8493
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