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The amino-terminal peptide of Bax perturbs intracellular Ca(2+) homeostasis to enhance apoptosis in prostate cancer cells
During apoptosis, proteolytic cleavage of Bax at the amino terminus generates a truncated Bax of ∼18 kDa (p18Bax) and an amino-terminal peptide of ∼3 kDa (p3Bax). Whereas extensive studies have shown that p18Bax behaves like a BH3 protein with enhanced pro-apoptotic function over that of the full-le...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Physiological Society
2009
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4116348/ https://ncbi.nlm.nih.gov/pubmed/19091958 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpcell.00390.2008 |
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