Prolonged Monoacylglycerol Lipase Blockade Causes Equivalent Cannabinoid Receptor Type 1 Receptor–Mediated Adaptations in Fatty Acid Amide Hydrolase Wild-Type and Knockout Mice

Complementary genetic and pharmacological approaches to inhibit monoacylglycerol lipase (MAGL) and fatty acid amide hydrolase (FAAH), the primary hydrolytic enzymes of the respective endogenous cannabinoids 2-arachidonoylglycerol (2-AG) and N-arachidonoylethanolamine, enable the exploration of poten...

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Detalhes bibliográficos
Main Authors: Schlosburg, Joel E., Kinsey, Steven G., Ignatowska-Jankowska, Bogna, Ramesh, Divya, Abdullah, Rehab A., Tao, Qing, Booker, Lamont, Long, Jonathan Z., Selley, Dana E., Cravatt, Benjamin F., Lichtman, Aron H.
Formato: Artigo
Idioma:Inglês
Publicado em: The American Society for Pharmacology and Experimental Therapeutics 2014
Assuntos:
Behavioral Pharmacology
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4109488/
https://ncbi.nlm.nih.gov/pubmed/24849924
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1124/jpet.114.212753
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