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Altered L-type Ca(2+) channel activity contributes to exacerbated hypoperfusion and mortality in smooth muscle cell BK channel-deficient septic mice
We determined the contribution of vascular large conductance Ca(2+)-activated K(+) (BK) and L-type Ca(2+) channel dysregulation to exaggerated mortality in cecal ligation/puncture (CLP)-induced septic BK channel β1-subunit knockout (BK β1-KO, smooth muscle specific) mice. CLP-induced hemodynamic cha...
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| Hoofdauteurs: | , , , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
American Physiological Society
2014
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4101616/ https://ncbi.nlm.nih.gov/pubmed/24829499 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpregu.00117.2014 |
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