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IL-18 induces profibrotic renal tubular cell injury via STAT3 activation
IL-18 is an important mediator of obstruction-induced renal fibrosis and renal tubular epithelial cell (TEC) injury. IL-18's proinflammatory properties have been attributed, in part, to NF-κB activation and the stimulation of cytokine gene expression; however, STAT3 has increasingly been shown...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Physiological Society
2013
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4073972/ https://ncbi.nlm.nih.gov/pubmed/23904224 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00620.2012 |
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