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Pancreatic β-cell Dedifferentiation in Diabetes and Re-differentiation following Insulin Therapy
Diabetes is characterized by ‘glucotoxic’ loss of pancreatic β-cell function and insulin content, but underlying mechanisms remain unclear. A mouse model of insulin-secretory deficiency induced by β-cell inexcitability (K(ATP) gain-of-function) demonstrates development of diabetes and reiterates the...
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| Hauptverfasser: | , , , |
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| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
2014
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4067979/ https://ncbi.nlm.nih.gov/pubmed/24746806 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cmet.2014.03.010 |
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