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Contact inhibition and high cell density deactivate the mammalian target of rapamycin pathway, thus suppressing the senescence program
During cell cycle arrest caused by contact inhibition (CI), cells do not undergo senescence, thus resuming proliferation after replating. The mechanism of senescence avoidance during CI is unknown. Recently, it was demonstrated that the senescence program, namely conversion from cell cycle arrest to...
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| Hoofdauteurs: | , , |
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| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
National Academy of Sciences
2014
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4066505/ https://ncbi.nlm.nih.gov/pubmed/24889617 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1405723111 |
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