Loss of UCP2 Attenuates Mitochondrial Dysfunction without Altering ROS Production and Uncoupling Activity

Although mitochondrial dysfunction is often accompanied by excessive reactive oxygen species (ROS) production, we previously showed that an increase in random somatic mtDNA mutations does not result in increased oxidative stress. Normal levels of ROS and oxidative stress could also be a result of an...

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Bibliografiske detaljer
Main Authors: Kukat, Alexandra, Dogan, Sukru Anil, Edgar, Daniel, Mourier, Arnaud, Jacoby, Christoph, Maiti, Priyanka, Mauer, Jan, Becker, Christina, Senft, Katharina, Wibom, Rolf, Kudin, Alexei P., Hultenby, Kjell, Flögel, Ulrich, Rosenkranz, Stephan, Ricquier, Daniel, Kunz, Wolfram S., Trifunovic, Aleksandra
Format: Artigo
Sprog:Inglês
Udgivet: Public Library of Science 2014
Fag:
Research Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4063685/
https://ncbi.nlm.nih.gov/pubmed/24945157
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pgen.1004385
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