Loss of UCP2 Attenuates Mitochondrial Dysfunction without Altering ROS Production and Uncoupling Activity

Although mitochondrial dysfunction is often accompanied by excessive reactive oxygen species (ROS) production, we previously showed that an increase in random somatic mtDNA mutations does not result in increased oxidative stress. Normal levels of ROS and oxidative stress could also be a result of an...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Kukat, Alexandra, Dogan, Sukru Anil, Edgar, Daniel, Mourier, Arnaud, Jacoby, Christoph, Maiti, Priyanka, Mauer, Jan, Becker, Christina, Senft, Katharina, Wibom, Rolf, Kudin, Alexei P., Hultenby, Kjell, Flögel, Ulrich, Rosenkranz, Stephan, Ricquier, Daniel, Kunz, Wolfram S., Trifunovic, Aleksandra
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Public Library of Science 2014
Aiheet:
Research Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4063685/
https://ncbi.nlm.nih.gov/pubmed/24945157
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pgen.1004385
Tagit: Lisää tagi
Ei tageja, Lisää ensimmäinen tagi!

Samankaltaisia teoksia