Sustained hyperoxia-induced NF-κB activation improves survival and preserves lung development in neonatal mice

Oxygen toxicity contributes to the pathogenesis of bronchopulmonary dysplasia (BPD). Neonatal mice exposed to hyperoxia develop a simplified lung structure that resembles BPD. Sustained activation of the transcription factor NF-κB and increased expression of protective target genes attenuate hyperox...

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Main Authors: McKenna, Sarah, Michaelis, Katherine A., Agboke, Fadeke, Liu, Thanh, Han, Kristie, Yang, Guang, Dennery, Phyllis A., Wright, Clyde J.
Formato: Artigo
Idioma:Inglês
Publicado: American Physiological Society 2014
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Articles
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC4060010/
https://ncbi.nlm.nih.gov/pubmed/24748603
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00001.2014
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