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Sustained hyperoxia-induced NF-κB activation improves survival and preserves lung development in neonatal mice
Oxygen toxicity contributes to the pathogenesis of bronchopulmonary dysplasia (BPD). Neonatal mice exposed to hyperoxia develop a simplified lung structure that resembles BPD. Sustained activation of the transcription factor NF-κB and increased expression of protective target genes attenuate hyperox...
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| Autori principali: | , , , , , , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
American Physiological Society
2014
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4060010/ https://ncbi.nlm.nih.gov/pubmed/24748603 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajplung.00001.2014 |
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