JNK interaction with Sab mediates ER stress induced inhibition of mitochondrial respiration and cell death

Our aim was to better understand the mechanism and importance of sustained c-Jun N-terminal kinase (JNK) activation in endoplasmic reticulum (ER) stress and effects of ER stress on mitochondria by determining the role of mitochondrial JNK binding protein, Sab. Tunicamycin or brefeldin A induced a ra...

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Hlavní autoři: Win, S, Than, T A, Fernandez-Checa, J C, Kaplowitz, N
Médium: Artigo
Jazyk:Inglês
Vydáno: Nature Publishing Group 2014
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Original Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC4040675/
https://ncbi.nlm.nih.gov/pubmed/24407242
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2013.522
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