Co-targeting the MAPK and PI3K/AKT/mTOR pathways in two genetically engineered mouse models of schwann cell tumors reduces tumor grade and multiplicity

Lignende værker

• Correction: Co-targeting the MAPK and PI3K/AKT/mTOR pathways in two genetically engineered mouse models of schwann cell tumors reduces tumor grade and multiplicity
  af: Watson, Adrienne L., et al.
  Udgivet: (2020)
• PTEN and NF1 inactivation in Schwann cells produces a severe phenotype in the peripheral nervous system that promotes the development and malignant progression of peripheral nerve sheath tumors
  af: Keng, Vincent W., et al.
  Udgivet: (2012)
• Canonical Wnt/β-catenin Signaling Drives Human Schwann Cell Transformation, Progression, and Tumor Maintenance
  af: Watson, Adrienne L., et al.
  Udgivet: (2013)
• Conditional Inactivation of Pten with EGFR Overexpression in Schwann Cells Models Sporadic MPNST
  af: Keng, Vincent W., et al.
  Udgivet: (2012)
• Interweaving the strands: β-Catenin, an HIV Co-Receptor, and Schwann Cell Tumors
  af: Largaespada, David, et al.
  Udgivet: (2013)