In vivo inhibition of the mitochondrial H(+)-ATP synthase in neurons promotes metabolic preconditioning

A key transducer in energy conservation and signaling cell death is the mitochondrial H(+)-ATP synthase. The expression of the ATPase inhibitory factor 1 (IF1) is a strategy used by cancer cells to inhibit the activity of the H(+)-ATP synthase to generate a ROS signal that switches on cellular progr...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Päätekijät: Formentini, Laura, Pereira, Marta P, Sánchez-Cenizo, Laura, Santacatterina, Fulvio, Lucas, José J, Navarro, Carmen, Martínez-Serrano, Alberto, Cuezva, José M
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Blackwell Publishing Ltd 2014
Aiheet:
Articles
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4000092/
https://ncbi.nlm.nih.gov/pubmed/24521670
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/embj.201386392
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