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Mitochondrial metals as a potential therapeutic target in neurodegeneration

Transition metals are critical for enzyme function and protein folding, but in excess can mediate neurotoxic oxidative processes. As mitochondria are particularly vulnerable to oxidative damage due to radicals generated during ATP production, mitochondrial biometal homeostasis must therefore be tigh...

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Bibliografski detalji
Glavni autori: Grubman, A, White, A R, Liddell, J R
Format: Artigo
Jezik:Inglês
Izdano: Blackwell Publishing Ltd 2014
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3976628/
https://ncbi.nlm.nih.gov/pubmed/24206195
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/bph.12513
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