Desmin modifications associate with amyloid-like oligomers deposition in heart failure

AIMS: The ultimate cause of heart failure (HF) is not known to date. The cytoskeletal protein desmin is differentially modified and forms amyloid-like oligomers in HF. We postulated that desmin post-translational modifications (PTMs) could drive aberrant desmin aggregation in HF. Therefore, we ident...

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Autori principali: Agnetti, Giulio, Halperin, Victoria L., Kirk, Jonathan A., Chakir, Khalid, Guo, Yurong, Lund, Linda, Nicolini, Francesco, Gherli, Tiziano, Guarnieri, Carlo, Caldarera, Claudio M., Tomaselli, Gordon F., Kass, David A., Van Eyk, Jennifer E.
Natura: Artigo
Lingua:Inglês
Pubblicazione: Oxford University Press 2014
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Original Articles
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3958618/
https://ncbi.nlm.nih.gov/pubmed/24413773
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/cvr/cvu003
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