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Loss of ATE1-mediated arginylation leads to impaired platelet myosin phosphorylation, clot retraction, and in vivo thrombosis formation
Protein arginylation by arginyl–transfer RNA protein transferase (ATE1) is emerging as a regulator protein function that is reminiscent of phosphorylation. For example, arginylation of β-actin has been found to regulate lamellipodial formation at the leading edge in fibroblasts. This finding suggest...
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| Autors principals: | , , , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Ferrata Storti Foundation
2014
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| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3943321/ https://ncbi.nlm.nih.gov/pubmed/24293517 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3324/haematol.2013.093047 |
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