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Hydrogen sulfide cytoprotective signaling is endothelial nitric oxide synthase-nitric oxide dependent

Previous studies have demonstrated that hydrogen sulfide (H(2)S) protects against multiple cardiovascular disease states in a similar manner as nitric oxide (NO). H(2)S therapy also has been shown to augment NO bioavailability and signaling. The purpose of this study was to investigate the impact of...

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Bibliografiska uppgifter
Huvudupphovsmän: King, Adrienne L., Polhemus, David J., Bhushan, Shashi, Otsuka, Hiroyuki, Kondo, Kazuhisa, Nicholson, Chad K., Bradley, Jessica M., Islam, Kazi N., Calvert, John W., Tao, Ya-Xiong, Dugas, Tammy R., Kelley, Eric E., Elrod, John W., Huang, Paul L., Wang, Rui, Lefer, David J.
Materialtyp: Artigo
Språk:Inglês
Publicerad: National Academy of Sciences 2014
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Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC3939925/
https://ncbi.nlm.nih.gov/pubmed/24516168
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1321871111
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