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Hijacking PrP(c)-dependent signal transduction: when prions impair Aβ clearance
The cellular prion protein PrP(c) is the normal counterpart of the scrapie prion protein PrP( Sc), the main component of the infectious agent of transmissible spongiform encephalopathies. The recent discovery that PrP( c) can serve as a receptor for the amyloid beta (Aβ) peptide and relay its neurot...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Frontiers Media S.A.
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3938157/ https://ncbi.nlm.nih.gov/pubmed/24592237 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fnagi.2014.00025 |
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