Hijacking PrP(c)-dependent signal transduction: when prions impair Aβ clearance

The cellular prion protein PrP(c) is the normal counterpart of the scrapie prion protein PrP( Sc), the main component of the infectious agent of transmissible spongiform encephalopathies. The recent discovery that PrP( c) can serve as a receptor for the amyloid beta (Aβ) peptide and relay its neurot...

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Autores principales: Hernandez-Rapp, Julia, Martin-Lannerée, Séverine, Hirsch, Théo Z., Launay, Jean-Marie, Mouillet-Richard, Sophie
Formato: Artigo
Lenguaje:Inglês
Publicado: Frontiers Media S.A. 2014
Materias:
Neuroscience
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC3938157/
https://ncbi.nlm.nih.gov/pubmed/24592237
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fnagi.2014.00025
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