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The Unfolded Protein Response Regulates Cardiac Sodium Current in Systolic Human Heart Failure

BACKGROUND: Human heart failure (HF) increases alternative mRNA splicing of the SCN5A cardiac Na(+) channel, generating variants encoding truncated, nonfunctional channels that are trapped in the endoplasmic reticulum. In this work, we tested whether truncated Na(+) channels activate the unfolded pr...

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Detalhes bibliográficos
Main Authors: Gao, Ge, Xie, An, Zhang, Jianhua, Herman, Amanda M., Jeong, Euy-Myoung, Gu, Lianzhi, Liu, Man, Yang, Kai-Chien, Kamp, Timothy J., Dudley, Samuel C.
Formato: Artigo
Idioma:Inglês
Publicado em: 2013
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3909518/
https://ncbi.nlm.nih.gov/pubmed/24036084
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCEP.113.000274
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