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Supplementation of nitric oxide attenuates AβPP and BACE1 protein in cerebral microcirculation of eNOS-deficient mice
Many cardiovascular risk factors, associated with an increased incidence of Alzheimer’s disease, are characterized by reduced bioavailability of nitric oxide (NO). Recently, we demonstrated in endothelial nitric oxide synthase deficient (eNOS(-/-)) mice loss of endothelial NO led to increased protei...
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| Autori principali: | , , |
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| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
2013
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3907936/ https://ncbi.nlm.nih.gov/pubmed/22886025 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3233/JAD-2012-121351 |
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