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JNK1 Protects against Glucolipotoxicity-Mediated Beta-Cell Apoptosis
Pancreatic β-cell dysfunction is central to type 2 diabetes pathogenesis. Prolonged elevated levels of circulating free-fatty acids and hyperglycemia, also termed glucolipotoxicity, mediate β-cell dysfunction and apoptosis associated with increased c-Jun N-terminal Kinase (JNK) activity. Endoplasmic...
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| 主要な著者: | , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Public Library of Science
2014
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3901710/ https://ncbi.nlm.nih.gov/pubmed/24475223 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0087067 |
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