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Aggregation-triggering segments of SOD1 fibril formation support a common pathway for familial and sporadic ALS
ALS is a terminal disease of motor neurons that is characterized by accumulation of proteinaceous deposits in affected cells. Pathological deposition of mutated Cu/Zn superoxide dismutase (SOD1) accounts for ∼20% of the familial ALS (fALS) cases. However, understanding the molecular link between mut...
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| Main Authors: | , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3890817/ https://ncbi.nlm.nih.gov/pubmed/24344300 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1320786110 |
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