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Chronic Angiotensin II infusion drives extensive aldosterone-independent ENaC activation
The inability of mineralocorticoid receptor (MR) blockade to reduce hypertension associated with high Angiotensin (Ang) II suggests direct actions of Ang II to regulate tubular sodium reabsorption via the epithelial Na(+) channel (ENaC) in the aldosterone-sensitive distal nephron (ASDN). We used fre...
Wedi'i Gadw mewn:
| Prif Awduron: | , , , , , , |
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| Fformat: | Artigo |
| Iaith: | Inglês |
| Cyhoeddwyd: |
2013
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| Pynciau: | |
| Mynediad Ar-lein: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3889165/ https://ncbi.nlm.nih.gov/pubmed/24060890 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/HYPERTENSIONAHA.113.01797 |
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