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Somatic inactivation of Tp53 in hematopoietic stem cells or thymocytes predisposes mice to thymic lymphomas with clonal translocations

TP53 protects cells from transformation by responding to stresses including aneuploidy and DNA double-strand breaks (DSBs). TP53 induces apoptosis of lymphocytes with persistent DSBs at antigen receptor loci and other genomic loci to prevent these lesions from generating oncogenic translocations. De...

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Detalhes bibliográficos
Main Authors: DeMicco, Amy, Yang-Iott, Katherine, Bassing, Craig H
Formato: Artigo
Idioma:Inglês
Publicado em: Landes Bioscience 2013
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3885641/
https://ncbi.nlm.nih.gov/pubmed/24036547
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4161/cc.26299
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