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Transgenic expression of survivin compensates for OX40-deficiency in driving Th2 development and allergic inflammation
Survivin, an inhibitor of apoptosis family molecule, has been proposed as a crucial intermediate in the signaling pathways leading to T-cell development, proliferation, and expansion. However, the importance of survivin to T-cell-driven inflammatory responses has not been demonstrated. Here, we show...
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| Autors principals: | , , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2013
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3876962/ https://ncbi.nlm.nih.gov/pubmed/23616302 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/eji.201243081 |
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