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Human LilrB2 Is a β-Amyloid Receptor and Its Murine Homolog PirB Regulates Synaptic Plasticity in an Alzheimer’s Model

Soluble β-amyloid (Aβ) oligomers impair synaptic plasticity and cause synaptic loss associated with Alzheimer’s disease (AD). We report that murine PirB (paired immunoglobulin-like receptor B) and its human ortholog LilrB2 (leukocyte immunoglobulin-like receptor B2), present in human brain, are rece...

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Bibliographische Detailangaben
Hauptverfasser: Kim, Taeho, Vidal, George S., Djurisic, Maja, William, Christopher M., Birnbaum, Michael E., Garcia, K. Christopher, Hyman, Bradley T., Shatz, Carla J.
Format: Artigo
Sprache:Inglês
Veröffentlicht: 2013
Schlagworte:
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3853120/
https://ncbi.nlm.nih.gov/pubmed/24052308
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1126/science.1242077
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