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The CD226/CD155 interaction regulates the pro-inflammatory (Th1/Th17)/ anti-inflammatory (Th2) balance in humans

CD226 costimulatory signals strongly promote Th1 differentiation enhancing IFN(γ) production by naïve T cells. We recently reported that knockdown of CD226 on human T cells resulted in a decrease in T-bet and IFN(γ) expression. However, the role of CD226 on Th2 and Th17 cells remains unknown. Here,...

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Autori principali: Lozano, Ester, Joller, Nicole, Cao, Yonghao, Kuchroo, Vijay, Hafler, David A.
Natura: Artigo
Lingua:Inglês
Pubblicazione: 2013
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC3819731/
https://ncbi.nlm.nih.gov/pubmed/23980210
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1300945
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