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IFNγ promotes complement expression and attenuates amyloid plaque deposition in APP transgenic mice
Reactive gliosis surrounding amyloid β (Aβ) plaques is an early feature of Alzheimer’s disease (AD) pathogenesis and may signify activation of the innate immune system in an attempt to clear or neutralize Aβ aggregates. In order to evaluate the role of IFNγ mediated neuroinflammation on the evolutio...
Kaydedildi:
| Asıl Yazarlar: | , , , , , |
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| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
2010
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3798002/ https://ncbi.nlm.nih.gov/pubmed/20368278 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.0903382 |
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