Cooperativity between MAPK and PI3K signaling activation is required for glioblastoma pathogenesis

BACKGROUND: Glioblastoma (GBM) genomes feature recurrent genetic alterations that dysregulate core intracellular signaling pathways, including the G1/S cell cycle checkpoint and the MAPK and PI3K effector arms of receptor tyrosine kinase (RTK) signaling. Elucidation of the phenotypic consequences of...

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Autors principals: Vitucci, Mark, Karpinich, Natalie O., Bash, Ryan E., Werneke, Andrea M., Schmid, Ralf S., White, Kristen K., McNeill, Robert S., Huff, Byron, Wang, Sophie, Van Dyke, Terry, Miller, C. Ryan
Format: Artigo
Idioma:Inglês
Publicat: Oxford University Press 2013
Matèries:
Basic and Translational Investigations
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3779038/
https://ncbi.nlm.nih.gov/pubmed/23814263
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/neuonc/not084
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