טוען...
Cooperativity between MAPK and PI3K signaling activation is required for glioblastoma pathogenesis
BACKGROUND: Glioblastoma (GBM) genomes feature recurrent genetic alterations that dysregulate core intracellular signaling pathways, including the G1/S cell cycle checkpoint and the MAPK and PI3K effector arms of receptor tyrosine kinase (RTK) signaling. Elucidation of the phenotypic consequences of...
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| Main Authors: | , , , , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Oxford University Press
2013
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3779038/ https://ncbi.nlm.nih.gov/pubmed/23814263 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/neuonc/not084 |
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