Oxygen radicals mediate endothelial cell damage by complement-stimulated granulocytes. An in vitro model of immune vascular damage.

During hemodialysis, alternative pathway complement activation leads to pulmonary sequestration of granulocytes, with loss of pulmonary vascular endothelial integrity and, at times, protein-rich pulmonary edema. An in vitro model of this phenomenon was constructed utilizing 51Cr-labeled human umbili...

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Dades bibliogràfiques

Publicat a:	J Clin Invest
Autors principals:	Sacks, T, Moldow, C F, Craddock, P R, Bowers, T K, Jacob, H S
Format:	Artigo
Idioma:	Inglês
Publicat:	American Society for Clinical Investigation 1978
Matèries:	Research Article
Accés en línia:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC372636/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/207729/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI109031
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