Oxygen radicals mediate endothelial cell damage by complement-stimulated granulocytes. An in vitro model of immune vascular damage.

During hemodialysis, alternative pathway complement activation leads to pulmonary sequestration of granulocytes, with loss of pulmonary vascular endothelial integrity and, at times, protein-rich pulmonary edema. An in vitro model of this phenomenon was constructed utilizing 51Cr-labeled human umbili...

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Bibliografiska uppgifter

I publikationen:	J Clin Invest
Huvudupphov:	Sacks, T, Moldow, C F, Craddock, P R, Bowers, T K, Jacob, H S
Materialtyp:	Artigo
Språk:	Inglês
Utgiven:	American Society for Clinical Investigation 1978
Ämnen:	Research Article
Länkar:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC372636/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/207729/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI109031
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