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Oxygen radicals mediate endothelial cell damage by complement-stimulated granulocytes. An in vitro model of immune vascular damage.

During hemodialysis, alternative pathway complement activation leads to pulmonary sequestration of granulocytes, with loss of pulmonary vascular endothelial integrity and, at times, protein-rich pulmonary edema. An in vitro model of this phenomenon was constructed utilizing 51Cr-labeled human umbili...

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Détails bibliographiques
Auteurs principaux: Sacks, T, Moldow, C F, Craddock, P R, Bowers, T K, Jacob, H S
Format: Artigo
Langue:Inglês
Publié: 1978
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC372636/
https://ncbi.nlm.nih.gov/pubmed/207729
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