Oxygen radicals mediate endothelial cell damage by complement-stimulated granulocytes. An in vitro model of immune vascular damage.

During hemodialysis, alternative pathway complement activation leads to pulmonary sequestration of granulocytes, with loss of pulmonary vascular endothelial integrity and, at times, protein-rich pulmonary edema. An in vitro model of this phenomenon was constructed utilizing 51Cr-labeled human umbili...

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Bibliografske podrobnosti

izdano v:	J Clin Invest
Principais autores:	Sacks, T, Moldow, C F, Craddock, P R, Bowers, T K, Jacob, H S
Format:	Artigo
Jezik:	Inglês
Izdano:	American Society for Clinical Investigation 1978
Teme:	Research Article
Online dostop:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC372636/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/207729/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI109031
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