Inhibition of prostacyclin by treatment of endothelium with aspirin. Correlation with platelet adherence.
Aspirin treatment of cultured endothelial cells from the umbilical vein increased the adherence of 51Cr-platelets when thrombin was present. If the cyclooxygenase activity of endothelium was inhibited by aspirin, as it is in the platelet, reduction of endogenous prostacyclin (PGI2) production could...
Guardat en:
| Publicat a: | J Clin Invest |
|---|---|
| Autors principals: | , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Society for Clinical Investigation
1979
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC372053/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/376548/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI109379 |
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