Inhibition of prostacyclin by treatment of endothelium with aspirin. Correlation with platelet adherence.
Aspirin treatment of cultured endothelial cells from the umbilical vein increased the adherence of 51Cr-platelets when thrombin was present. If the cyclooxygenase activity of endothelium was inhibited by aspirin, as it is in the platelet, reduction of endogenous prostacyclin (PGI2) production could...
Gorde:
| Argitaratua izan da: | J Clin Invest |
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| Egile Nagusiak: | , , , , |
| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
American Society for Clinical Investigation
1979
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC372053/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/376548/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI109379 |
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