Inhibition of prostacyclin by treatment of endothelium with aspirin. Correlation with platelet adherence.

Aspirin treatment of cultured endothelial cells from the umbilical vein increased the adherence of 51Cr-platelets when thrombin was present. If the cyclooxygenase activity of endothelium was inhibited by aspirin, as it is in the platelet, reduction of endogenous prostacyclin (PGI2) production could...

Celý popis

Podrobná bibliografie

Vydáno v:	J Clin Invest
Hlavní autoři:	Czervionke, R L, Smith, J B, Fry, G L, Hoak, J C, Haycraft, D L
Médium:	Artigo
Jazyk:	Inglês
Vydáno:	American Society for Clinical Investigation 1979
Témata:	Research Article
On-line přístup:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC372053/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/376548/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI109379
Tagy:	Přidat tag Žádné tagy, Buďte první, kdo vytvoří štítek k tomuto záznamu!