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Proliferation of amyloid-β42 aggregates occurs through a secondary nucleation mechanism
The generation of toxic oligomers during the aggregation of the amyloid-β (Aβ) peptide Aβ42 into amyloid fibrils and plaques has emerged as a central feature of the onset and progression of Alzheimer’s disease, but the molecular pathways that control pathological aggregation have proved challenging...
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| Hlavní autoři: | , , , , , , , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
National Academy of Sciences
2013
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3683769/ https://ncbi.nlm.nih.gov/pubmed/23703910 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1218402110 |
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