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Letm1, the mitochondrial Ca(2+)/H(+) antiporter, is essential for normal glucose metabolism and alters brain function in Wolf–Hirschhorn syndrome
Mitochondrial metabolism, respiration, and ATP production necessitate ion transport across the inner mitochondrial membrane. Leucine zipper-EF-hand containing transmembrane protein 1 (Letm1), one of the genes deleted in Wolf–Hirschhorn syndrome, encodes a putative mitochondrial Ca(2+)/H(+) antiporte...
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| Main Authors: | , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
National Academy of Sciences
2013
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3683736/ https://ncbi.nlm.nih.gov/pubmed/23716663 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1308558110 |
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