Attenuation of inflammatory mediator production by the NF-κB member RelB is mediated by microRNA-146a in lung fibroblasts

Podobne knjige/članki

• Lung-Targeted Overexpression of the NF-κB Member RelB Inhibits Cigarette Smoke–Induced Inflammation
  od: McMillan, David H., et al.
  Izdano: (2011)
• The Aryl Hydrocarbon Receptor Attenuates Tobacco Smoke-induced Cyclooxygenase-2 and Prostaglandin Production in Lung Fibroblasts through Regulation of the NF-κB Family Member RelB
  od: Baglole, Carolyn J., et al.
  Izdano: (2008)
• Aryl Hydrocarbon Receptor-Deficient Mice Develop Heightened Inflammatory Responses to Cigarette Smoke and Endotoxin Associated with Rapid Loss of the Nuclear Factor-κB Component RelB
  od: Thatcher, Thomas H., et al.
  Izdano: (2007)
• B-cell survival and development controlled by the coordination of NF-κB family members RelB and cRel
  od: Almaden, Jonathan V., et al.
  Izdano: (2016)
• RelB and Neuroinflammation
  od: Mockenhaupt, Karli, et al.
  Izdano: (2021)