EGFR mutation-induced alternative splicing of Max contributes to growth of glycolytic tumors in brain cancer

Alternative splicing contributes to diverse aspects of cancer pathogenesis including altered cellular metabolism, but the specificity of the process or its consequences are not well understood. We characterized genome-wide alternative splicing induced by the activating EGFRvIII mutation in glioblast...

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Bibliografiset tiedot
Päätekijät: Babic, Ivan, Anderson, Erik S., Tanaka, Kazuhiro, Guo, Deliang, Masui, Kenta, Li, Bing, Zhu, Shaojun, Gu, Yuchao, Villa, Genaro, Akhavan, David, Nathanson, David, Gini, Beatrice, Mareninov, Sergey, Li, Rui, Espindola C., Carolina, Kurdistani, Siavash K., Eskin, Ascia, Nelson, Stanley F., Yong, William H., Cavenee, Webster K., Cloughesy, Timothy F., Christofk, Heather R., Black, Douglas L., Mischel, Paul S.
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2013
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Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC3679227/
https://ncbi.nlm.nih.gov/pubmed/23707073
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cmet.2013.04.013
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