EGFR mutation-induced alternative splicing of Max contributes to growth of glycolytic tumors in brain cancer

Alternative splicing contributes to diverse aspects of cancer pathogenesis including altered cellular metabolism, but the specificity of the process or its consequences are not well understood. We characterized genome-wide alternative splicing induced by the activating EGFRvIII mutation in glioblast...

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Main Authors: Babic, Ivan, Anderson, Erik S., Tanaka, Kazuhiro, Guo, Deliang, Masui, Kenta, Li, Bing, Zhu, Shaojun, Gu, Yuchao, Villa, Genaro, Akhavan, David, Nathanson, David, Gini, Beatrice, Mareninov, Sergey, Li, Rui, Espindola C., Carolina, Kurdistani, Siavash K., Eskin, Ascia, Nelson, Stanley F., Yong, William H., Cavenee, Webster K., Cloughesy, Timothy F., Christofk, Heather R., Black, Douglas L., Mischel, Paul S.
Format: Artigo
Jezik:Inglês
Izdano: 2013
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Article
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC3679227/
https://ncbi.nlm.nih.gov/pubmed/23707073
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cmet.2013.04.013
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